+AAV在(zai)肝臟研究中的靶向策略【應用篇】
+AAV在(zai)胰腺研究中的(de)靶(ba)向策(ce)略(干(gan)貨&應用合集(ji))
+AAV在腸道中的靶(ba)向策略(lve)
+『糖尿病(bing)治療(liao)新(xin)靶點(dian)』南京大學韓(han)曉教授團(tuan)隊發現HRD1——T2D治療(liao)新(xin)靶點(dian)!
+案(an)例分享 ‖ AAV9感(gan)染腸道(dao)組織
+《Molecular Cell》‖ 維真AAV與自噬研究和脂代謝
+維真AAV感(gan)染腸道(dao)案例分享
+PCSK9與動脈(mo)粥樣硬化
+維真生(sheng)物AAV8感染(ran)小鼠胰(yi)腺
肝臟方面(mian):
1.
Proc. Natl. Acad. Sci. U.S.A. (PNAS). (IF=9.412). Zhang, et.al. (2020). Hepatic neddylation targets and stabilizes electron transfer flavoproteins to facilitate fatty acid β-oxidation.[中國人民解放軍軍事醫學科學院 & 南開大學 AAV-DJ-CAG-Cre II型戊二酸尿癥GA-II]
2.
Journal of Hepatology. (IF=20.582). She, et.al. (2019). PSMP/MSMP promotes hepatic ?brosis through CCR2 and represents a novel therapeutic target.[北京大學基礎醫學院 AAV8-hPSMP & null 肝纖維化]
3.
Theranostics. (IF=8.579). Liu, et.al. (2019). Suppression of YAP/TAZ-Notch1-NICD axis by bromodomain and extraterminal protein inhibition impairs liver regeneration.[浙江大學 AAV9-CMV-YAP 肝再生]
4.
Molecular Cell. (IF=15.584). Wan, et.al. (2019). Pacer is a mediator of mTORC1 and GSK3-TIP60 signaling in regulation of autophagosome maturation and lipid metabolism.[浙江大學 AAV9-mCherry-GFP-LC3 and AAV9-Pacerwt-HA&Pacer2KR-HA&PacerS157A-HA&PacerS157D-HA 肝自噬和脂代謝]
5.
Experimental Cell Research. (IF=3.383). Li, et.al. (2018). Brg1 promotes liver fibrosis via activation of hepatic stellate cells.[華中科技大學同濟醫學院附屬同濟醫院 AAV8-shBrg1 or AAV8-GFP 肝纖維化]
6.
Molecular Cell. (IF=15.584). Wan, et.al. (2018). mTORC1-Regulated and HUWE1-Mediated WIPI2 Degradation Controls Autophagy Flux.[浙江大學 AAV9-shHUWE1 & shNC and AAV9-WIPI2&WIPI2-S395A&WIPI2-S395D 肝自噬]
7.
Molecular Cell. (IF=15.584). Su, et.al. (2017). VPS34 Acetylation Controls Its Lipid Kinase Activity and the Initiation of Canonical and Non-canonical Autophagy.[浙江大學 AAV9-CMV-VPS34&3KR&3KQ 肝自噬]
8.
Proc. Natl. Acad. Sci. U.S.A. (PNAS). (IF=9.412). He, et.al. (2017). MicroRNA-351 promotes schistosomiasis-induced hepatic fibrosis by targeting the vitamin D receptor.[第二軍醫大學 AAV8-CMV-eGFP-miR-351-5p Sponge 血吸蟲病肝纖維化]
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